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University of the Sciences in Philadelphia
Department of Biomedical Writing
Regulatory Writing Certificate
Classes:
BW 703 Information Strategies for Biomedical Writers
BW 704 Regulatory Documentation Processes
BW 705 Biostatistics for Biomedical Writers
BW 708 Regulatory Writing: New Drug Applications
Marketing Writing Certificate
Classes:
BW 701 Professional Writing in Science
BW 702 Stylistics and Editing (Spring 2011)
BW 733 Health Journalism Documentation
BW 760 Entrepreneurship in Biomedical Communication (Spring 2011)
University of Illinois at Chicago
Department of Pharmacology
Ph.D. Pharmacology
Research Summary:
Under Chinnaswamy Tiruppathi, PhD, I studied the regulation of the calcium channel TRPC1 in
endothelial cells. When there is increased calcium inside endothelial cells, the cells contract and
increase the endothelium’s permeability. I found that TRPC1 was inhibited by a peptide based on the
sequence of the caveolin-1 scaffolding domain. The development of such an inhibitor can help treat
diseases such as septic shock and acute lung injury.
Classes:
PHYB 401 Human Physiology I
PHYB 402 Human Physiology II
PCOL 425 Medical Pharmacology
PCOL 505 Receptor Mechanisms
PCOL 530 Vascular Biology
BCHE 460 Biochemistry
BCHE 520 Biochemistry Research Techniques
GC 401 Scientific Integrity
Department of Biomedical Writing
Regulatory Writing Certificate
Classes:
BW 703 Information Strategies for Biomedical Writers
BW 704 Regulatory Documentation Processes
BW 705 Biostatistics for Biomedical Writers
BW 708 Regulatory Writing: New Drug Applications
Marketing Writing Certificate
Classes:
BW 701 Professional Writing in Science
BW 702 Stylistics and Editing (Spring 2011)
BW 733 Health Journalism Documentation
BW 760 Entrepreneurship in Biomedical Communication (Spring 2011)
University of Illinois at Chicago
Department of Pharmacology
Ph.D. Pharmacology
Research Summary:
Under Chinnaswamy Tiruppathi, PhD, I studied the regulation of the calcium channel TRPC1 in
endothelial cells. When there is increased calcium inside endothelial cells, the cells contract and
increase the endothelium’s permeability. I found that TRPC1 was inhibited by a peptide based on the
sequence of the caveolin-1 scaffolding domain. The development of such an inhibitor can help treat
diseases such as septic shock and acute lung injury.
Classes:
PHYB 401 Human Physiology I
PHYB 402 Human Physiology II
PCOL 425 Medical Pharmacology
PCOL 505 Receptor Mechanisms
PCOL 530 Vascular Biology
BCHE 460 Biochemistry
BCHE 520 Biochemistry Research Techniques
GC 401 Scientific Integrity